Neidhart and
Matthias conclude that the origin of this condition might be
sought for in the disturbance of the nerve-centers. After
reviewing all these theories, Salzer states that in his case the
patient was previously healthy and never had suffered the
slightest hemorrhage in any part, and he therefore rejects the
theory of extravasation. He is inclined to suppose that the
priapism was due to the stimulation of the nervi erigentes,
brought about either by anatomic change in the nerves themselves,
or by pressure upon them by enlarged lumbar glands, an associate
condition of leukemia.
Burchard reports a most interesting case of prolonged priapism in
an English gentleman of fifty-three. When he was called to see
the man on July 15th he found him suffering with intense pain in
the penis, and in a state of extreme exhaustion after an erection
which had lasted five hours uninterruptedly, during the whole of
which time the organ was in a state of violent and continuous
spasm. The paroxysm was controlled by 3/4 grain morphin and 1/50
grain atropin. Five hours later, after a troubled sleep, there
was another erection, which was again relieved by hypodermic
medication. During the day he had two other paroxysms, one
lasting forty-five minutes; and another, three hours later,
lasting eighteen minutes.
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