On his return to England his penis was flaccid, and
his symptoms had disappeared.
Salzer presents an interesting paper on priapism which was quoted
in The Practitioner of London. Salzer describes one patient of
forty-six who awoke one morning with a strong erection that could
not be reduced by any means. Urine was voided by jerks and with
difficulty, and only when the subject was placed in the knee and
elbow position. Despite all treatment this condition continued
for seven weeks. At this time the patient's spleen was noticed to
be enormously enlarged. The man died about a year after the
attack, but a necropsy was unfortunately refused. Salzer, in
discussing the theories of priapism, mentions eight cases
previously reported, and concludes, that such cases are
attributable to leukemia. Kremine believes that continued
priapism is produced by effusion of blood into the corpora
cavernosa, which is impeded on its return. He thinks it
corresponds to bleeding at the nose and rectum, which often
occurs in perfectly healthy persons. Longuet regards the
condition of the blood in leukemia as the cause of such priapism,
and considers that the circulation of the blood is retarded in
the smaller vessels, while, owing to the great increase in the
number of white corpuscles, thrombi are formed.
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